Case report: Reversible posterior leukoencephalopathy in a venomous snake (Bothrops asper) bite victim

Miguel E. Delgado; Oscar H. Del Brutto

doi:10.4269/ajtmh.2012.11-0610

Case report: Reversible posterior leukoencephalopathy in a venomous snake (Bothrops asper) bite victim

Miguel E. Delgado, Oscar H. Del Brutto

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11 Citas (Scopus)

Resumen

An 18-year-old man developed posterior reversible leukoencephalopaty after being bitten by a venomous snake (Bothrops asper). It is possible that this previously unrecognized neurological complication of snake bite envenoming occurred as the result of endothelial dysfunction induced by the venom of the offending snake. This pathogenetic mechanism has also been implicated as the cause of cerebral infarctions in snake bite victims. Alternatively, the leukoencephalopathy might have been a complication of antivenom therapy.

Idioma original	Inglés
Páginas (desde-hasta)	496-498
Número de páginas	3
Publicación	American Journal of Tropical Medicine and Hygiene
Volumen	86
N.º	3
DOI	https://doi.org/10.4269/ajtmh.2012.11-0610
Estado	Publicada - mar. 2012
Publicado de forma externa	Sí

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title = "Case report: Reversible posterior leukoencephalopathy in a venomous snake (Bothrops asper) bite victim",

abstract = "An 18-year-old man developed posterior reversible leukoencephalopaty after being bitten by a venomous snake (Bothrops asper). It is possible that this previously unrecognized neurological complication of snake bite envenoming occurred as the result of endothelial dysfunction induced by the venom of the offending snake. This pathogenetic mechanism has also been implicated as the cause of cerebral infarctions in snake bite victims. Alternatively, the leukoencephalopathy might have been a complication of antivenom therapy.",

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AB - An 18-year-old man developed posterior reversible leukoencephalopaty after being bitten by a venomous snake (Bothrops asper). It is possible that this previously unrecognized neurological complication of snake bite envenoming occurred as the result of endothelial dysfunction induced by the venom of the offending snake. This pathogenetic mechanism has also been implicated as the cause of cerebral infarctions in snake bite victims. Alternatively, the leukoencephalopathy might have been a complication of antivenom therapy.

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Case report: Reversible posterior leukoencephalopathy in a venomous snake (Bothrops asper) bite victim

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